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MUSCLE GROWTHResearch OnlyGROWTH HORMONEGHRPANTI-AGING

Ipamorelin

Also known as: NNC 26-0161

27.6k views/week 421 citations 0 edits Updated 4/6/2026

Ipamorelin is a selective pentapeptide growth hormone releasing peptide (GHRP) that stimulates pituitary GH release with minimal impact on cortisol, prolactin, or ACTH levels. Its receptor selectivity and clean side-effect profile make it the most widely studied GHRP for anti-aging and recovery applications.

STRUCTURE

Molecular Composition

FORMULA
C₃₈H₄₉N₉O₅
MOL. WEIGHT
711.86 Da
SEQUENCE LENGTH
5 amino acids
CAS NUMBER
170851-70-4
HALF-LIFE
~2 hours (SQ)
SELECTIVITY
GHS-R1a only
AMINO ACID CHAIN VISUALIZATION
A
Aib (α-aminoisobutyric)
DPP-IV resistance
NH-CO
H
Histidine
GHS-R1a recognition
NH-CO
D
D-2-Nal
core pharmacophore
NH-CO
P
D-Phe-Ala
receptor selectivity
NH-CO
K
Lys-NH₂
C-terminal amide
SEQUENCEA-H-D-P-K
MECHANISMS

How It Works

🎯
Selective GHS-R1a Agonism
Ipamorelin binds GHS-R1a (the ghrelin receptor) on pituitary somatotrophs with high selectivity. Unlike GHRP-6 and GHRP-2, it does not meaningfully activate receptors for ACTH or prolactin release, making it the cleanest GHRP available. GHS-R1a activation triggers intracellular calcium signaling → GH vesicle release.
Synergy with GHRH
GHRH (via GHRHR) and ghrelin (via GHS-R1a) activate separate intracellular pathways (cAMP and IP3/Ca²⁺ respectively) that converge on GH secretion. Co-administration produces multiplicative GH release — studies show 10× more GH than either compound alone. This is the pharmacological basis of the ipamorelin + CJC-1295 stack.
🛡
No Cortisol / Prolactin Stimulation
Ipamorelin's selectivity for GHS-R1a over other GPCRs means it does not significantly elevate cortisol or prolactin — a critical advantage over GHRP-6 (which increases appetite and cortisol significantly) and GHRP-2 (which increases cortisol, prolactin, and ACTH). This makes ipamorelin superior for long-term protocols and stress-sensitive individuals.
📈
IGF-1 Upregulation
The GH pulses triggered by ipamorelin drive hepatic IGF-1 synthesis. IGF-1 mediates the downstream anabolic effects — skeletal muscle protein synthesis via PI3K/Akt/mTOR, nitrogen retention, anti-catabolism, collagen synthesis, and fat mobilisation. IGF-1 levels typically rise 20–40% above baseline after 4+ weeks of ipamorelin use.
OVERVIEW

Research Overview

Ipamorelin was developed by Novo Nordisk in the late 1990s as a highly selective ghrelin receptor (GHS-R1a) agonist. Its key differentiator from other GHRPs (GHRP-2, GHRP-6, Hexarelin) is selectivity: it robustly stimulates GH release without the corresponding cortisol, prolactin, or ACTH spikes seen with older GHRPs, which can suppress immune function, promote fat storage, and cause hormonal dysregulation.

Ipamorelin is commonly combined with CJC-1295, a GHRH analog, creating a synergistic dual-pathway approach — Ipamorelin triggers the amplitude of GH pulses while CJC-1295 increases pulse frequency. This combination mimics youthful GH secretory patterns more closely than either peptide alone.

Mechanism of Action

// SELECTIVE GHS-R1a AGONISM

Ipamorelin binds to the growth hormone secretagogue receptor 1a (GHS-R1a) on pituitary somatotrophs, triggering intracellular calcium influx and GH granule exocytosis. Unlike GHRP-6 and GHRP-2, Ipamorelin does not significantly activate GHS-R subtypes associated with cortisol or prolactin release.

// PULSATILE GH STIMULATION

GH release from Ipamorelin is pulsatile rather than tonic, mimicking the natural ultradian rhythm of GH secretion. Pulsatile release is important for preserving GH receptor sensitivity and avoiding the receptor downregulation that occurs with continuous GH administration.

SEQUENCE

Amino Acid Sequence

Aib-His-D-2-Nal-D-Phe-Lys-NH2
DOSAGE

Dosage & Administration

INJECTABLE (SUBCUTANEOUS) — STANDALONE
DOSE
200–300 µg
FREQUENCY
2–3× daily (before meals + at bedtime)
NOTES
Standalone protocol. Administer on an empty stomach — food blunts GH release. Inject 30–45 min before meals or 30–45 min before sleep to coincide with natural GH pulses. Rotate injection sites (abdomen/thigh).
INJECTABLE (SUBCUTANEOUS) — STACKED WITH CJC-1295 (NO DAC)
DOSE
100–200 µg ipamorelin + 100 µg CJC-1295 (no DAC)
FREQUENCY
2–3× daily; always together in the same injection
NOTES
Gold-standard combination protocol. Mix in the same syringe for simultaneous administration. The synergy between GHRH (CJC) and GHRP (ipamorelin) produces 10× greater GH release than either agent alone. Pre-workout and/or bedtime are the most impactful injection windows.

Ipamorelin is the most selective GHRP available — it releases GH without significant co-stimulation of cortisol, prolactin, or ACTH, which is the primary limitation of older GHRPs like GHRP-6. Its 5-amino-acid pentapeptide structure confers excellent bioavailability and tolerability. Side effects are minimal: mild transient water retention and flushing at higher doses. No desensitisation observed at standard doses in research. Always source from reputable suppliers with mass spec or HPLC certificate.

CYCLING

Cycle Duration Guide

ON CYCLE
8–12 weeks
OFF CYCLE
4–6 weeks minimum

Ipamorelin cycles are typically 8–12 weeks when used for body composition or recovery goals, followed by an off-cycle to allow pituitary sensitivity to reset. When stacked with CJC-1295 (no DAC), the same cycling principle applies to both peptides simultaneously. IGF-1 levels can be monitored mid-cycle as a proxy for GH axis activity.

Quick Reference
FORMULAC₃₈H₄₉N₉O₅
MOL. WEIGHT711.86 Da
LENGTH5 amino acids
ORIGINSynthetic pentapeptide; developed by Novo Nordisk
HALF-LIFE~2 hours (subcutaneous)
SOLUBILITYSoluble in bacteriostatic water or sterile saline
CAS NO.170851-70-4
STATUSResearch Only
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TAGS
growth hormoneGHRPanti-agingmuscle recoveryselectivefat loss